الفهرس 
Introduction and Aim of the WorkOnly 14 pages are availabe for public view
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Abstract
Aim of the work
To study changes in serum lipids and lipoproteins particularly lipoprotein (a) in cigarette smokers compared to non-smokers.
Conclusion
1- Cigarette smoking affects adversely the lipid profile leading to increased total cholesterol, LDL-C, triglyceride and decreased HDL-C.
2- Heavy smoking has no effect on Lp (a) which may be to a large extent genetically determined.
3- Moderate smoking may lead to decreased Lp (a), but the statistical power of this entity is low and large prospective study is needed.
4- We advise smokers to avoid and stop this habit immediately as the dyslipidaemic effect caused by smoking increases atherosclerosis. They also deprive from the antiatherogenic effect of HDL.
5- The smoker may, genetically have high Lp (a) which if more than 30 mg/dL the risk for myocardial infarction increases twofold and if his serum LDL level rises as a result of smoking, hence both factors are elevated and the risk of acquiring myocardial infarction is increased fivefold.