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Abstract
Physicians often find it problematic to manage patients with hair loss because of the difficulty in making a specific diagnosis and the small therapeutic spectrum currently available. The diagnosis ofhail JOSS is usually uabz,u”examination including identifying the pattern of hair loss and examination of the scalp. In some patients, hair pull test, daily hair count, microscopic hair analysis, appropriate diagnostic tests or punch biopsy may be necessary. These items are usually sufficient to categorize the form of hair disorder and provide a clue to the nature of the underlying disorder.Disease states that cause large numbers of hair to full outmay be classified into cicatricial alopecia and non-cicatricialalopecia. The non-cicatricial alopecia may be further classified intofocal (patterned) non-cicatricial alopecia or diffuse non-cicatricials.
alopecia.
The diffuse non-cicatricial hair loss occurs if a significant large number of these anagen hairs at one time are insulted by any geileralized physical or metabolic insult. This insult, whether systemic illness, nutritional deprivation, hormonal disturbance ,exposure to certain chemicals, toxicity from a drug or any other generalized cause, can affect the rapidly growing and proliferatingmatrix cells of anagen hair follicles. There are two potential responses of anagen hair follicle to these systemic insults. First response is to prematurely stop growing and terminating .anagen phase to move into catagen then telogen phase, which result clinically in the common condition termed telogen effluvium. The second response is to temporarily interrupt or slow the normal cell division and differentiation of matrix cells in the anagen which result clinically in less common condition termed anagen effluvium .Prognosis for hair regrowth is generally good if the cause can he found and eliminated and if the patient does not have associated pattern hair loss.