الفهرس 
Vitreous AnatomyOnly 14 pages are availabe for public view
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Abstract
Vitreous is the largest single tissue of the eye, but it is also the least energy demanding. Vitreous collagen fibrils are oriented randomly to form an irregular mesh in which hyaluronic acid molecules are trapped and are inserted into the basement membrane of the Muller and glial cells of the retina.
Stickler syndrome, a dominantly inherited disease caused by collagen gene mutation, affects the vitreous causing the vitreous to be membranous or empty.
A variety of unrelated disorders affect the vitreous throughout life. Common degenerative conditions due to aging (e.g., vitreous syneresis) place everyone at some risk for PVD, retinal tear, and retinal detachment.
The three most common causes of vitreous hemorrhage are proliferative diabetic retinopathy, posterior vitreous detachment (PVD) with or without retinal tears, and ocular trauma. The mechanisms of vitreous hemorrhage fall into three main categories: Abnormal vessels that are prone to bleeding, normal vessels that rupture under stress, or extension of blood from an adjacent source. Vitreous can loose its transparency from the accumulation of phospholipids (asteroid hyalosis), or cholesterol (synchesis scintillans).
Primary infectious and inflammatory disorders of the vitreous were summarized as follows: Pars planitis, Juvenile rheumatoid arthritis, Toxocariasis, Sarcoidosis, Lyme disease, Inflammatory bowel disease and Whipples disease.The vitreous is often the site of overflow, or secondary, inflammation from primary uveitic disorders.
Primary ocular lymphoma must always be considered in the differential diagnosis of chronic vitritis.
Vitreous membranes are the final common pathway for a number of different types of ocular injury. Their pathogenesis likely differs according to underlying cause, they share many features with the nonspecific inflammatory processes that promote wound healing.