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Abstract
ThE wide use of the organophosphorus pesticides (OF)
in pest control is one of the main causes of environmental
pollution. This class of compounds had the
potential for causing an acute toxicity, indicated by
signs of cholinergic poisoning, and a delayed neuropathy
that may develop days to weeks after exposure o~
some animal species, including humans, to certain organophosphorus
compounds (Davis and Rishardson, 1980). The
adult chicken has been the experimental species of
choice to study organophosphorus ester induced delayed
neurotoxicity since it exhibits clinical and histopathological
manifestations similar to those in humans (EPA,
1978). All of the OP insecticides can cause acute
cholinergic poisoning with temporary muscle weakness,
and few of these compounds have been shown to produce a
more persistent effect-delayed neurotoxicity, Organophosphorus
Compounds Induced Delayed Neurotoxicity (OPIDN)