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Abstract Background: Infections are leading cause of death in patients with acute stroke. Recent studies indicate that stroke leads to changes in the immune system which predisposed to infection. Aim of work: The objectives of this study are to define the contribution of specific populations of lymphocytes; CD4+CD25 (T-regulatory cells) lymphocytes; to post-stroke infection, their relation to degree of neurological deficit associated with stroke. Patients and Methods: Twenty-five patients with acute ischemic stroke subjected to clinical assessment and NIHSS score and flowcytometric analysis for T- |