الفهرس 
AcknowledgementOnly 14 pages are availabe for public view
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Abstract
Bariatric surgery has been performed for many years to achieve sustained
weight loss in the morbidly obese population. As a secondary effect, a remarkable improvement in glycemic control is commonly achieved postoperatively. This hasled to substantial interest in the use of bariatric procedures to treat type 2 diabetes.
In a minority of patients who have undergone gastric bypass, the raised
blood glucose associated with diabetes is not only resolved, but is actually
significantly decreased to result in hyperinsulinaemic hypoglycaemia.
Obesity effect male and female sex hormones and sexual function. Bariatric
surgery is associated with improved male sex hormone parameters.
It is well known that calcium and vitamin D deficiency can occur in patients
undergoing bariatric surgery. Malabsorption of calcium and vitamin D occurs from bypassing segments of intestine during bariatric operations. The malabsorption of
vitamin D contributes further to calcium malabsorption. With a relative lack of
calcium, the production of parathyroid hormone (PTH) is increased, which leads torelease of calcium from bone, potentially causing bone loss and long-term risk of
osteoporosis.
Pathological weight gain, termed hypothalamic obesity (HyOb), is often
severe, refractory to therapy, and has a significant negative impact on the quality of life for patients with Craniopharyngioma. The effect of bariatric surgery in this group of patient is debated and still under investigation.