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Abstract Polycystic ovary syndrome (PCOS) is a heterogeneous disorder which has eluded definitive description because of the varied combination of clinical, biochemical and ultrasonographic features which may occur. The commonest association is of hyperandrogenism and chronic anovulation; recognition of characteristic ovarian ultrasound features together with clinical symptoms of oligomenorrhoea, hyperandrogenism, infertility or obesity is presently the preferred approach to diagnosis (Trivax and Azziz, 2007). Polycystic ovary disease is one of the most common endocrine disorders, although its etiology remains unknown. At a joint consensus meeting of the American Society of Reproductive medicine and the European Society of Human Reproduction and Embryology held in Rotterdam, in May 2003, a refined definition of the polycystic ovary disease was agreed. Namely the presence of two out of the following three criteria: (i) oligo- and/or anovulation; (ii) hyper-androginism (clinical and/or biochemical); and (iii) polycystic ovaries, with the exclusion of other etiologies. The morphology of the polycystic ovary has been redefined as an ovary with 12 or more follicles measuring 2-9 mm in diameter and/or increased ovarian volume more than 10 ml (Balen et al., 2005). Transvaginal color and pulsed Doppler ultrasound in combination with B-mode imaging is used as a non-invasive method to assess blood flow in both obstetrics and gynecology this technique has been used to study the hemodynamic changes in the uterine and/or ovarian arteries during the menstrual cycle in women with normal ovaries. (Lakhani et al., 2002 ). There has been much interest regarding the potential role of transvaginal color and pulsed Doppler ultrasound in assessing the ovarian and uterine blood flow of PCOS (Dolz et al., 1999). Most investigators would agree that the blood flow and the vascular pattern of an organ are directly related to the organ’s morphology and function (Collins et al., 1991). |