الفهرس 
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Abstract
Sir charles Bell C 1833 ) first discovered that the facial musculature is innervated by a cranial nerve separate from the trigeminal nerve.
In 1893 Gowers noted that exposure to cold sometines preceäes the onset of facial palsy and ascribed it to neu— ntis of the facial nerve.
Peripheral facial palsy of undetermined etiology is now referred to as Bell’s palsy, and little has been added regarding its etiology or pethology since Gowers report.
C Schuknecht 1974 ).
Facial paralysis is by far the most common of all peripheral nerve lesions. Its effects upon voluntary and emotional facial expressions upon the mechanisms of mastication and speech, upon the sense of taste, and upon the protective mechanisms of the eye are grave enough in thems elves.
Added to the often gross cosniotic deformity these constitute a major calamity in the life of the sufferer and those near to him.
Otologists have the care of , and the responsibility
for , this orn1a1 nerve and major contributions in this field have been made by otologica]. pionears . C Groves 1979)
The etiology of Beil’s palsy , like a number of other cranial nerve affictions which consist of rapid or sudden loss of function , has never been satisfactorily explained despite extensive analysis by a number of investigators over the years ( Jannetta , 1978 ),
In this thesis we will discuss a.natony of the facial nerve , pathology of peripheral nerve lesions including degeneration and regeneration , etiology , pathogenesis of Bell’s palsy and inanagment including general lines , medical and surgical treatment.