الفهرس 
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Abstract
Acute myocardial infarction is usually caused by occlusive
coronary thrombosis initiated by rupture of an atheromatous
plaque. The subendocardium infarcts early after the occlusion, but
outward extension to affect the full thickness of the ventricular
wall may take several hours. Restoration of normal coronary flow,
before the transmural spread of infarction is complete, is now seen
as the primary goal of hospital treatment because it allows
reperfusion of the threatened myocardium with reduction of
eventual infarct size