الفهرس 
): Portal HypertensionOnly 14 pages are availabe for public view
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Abstract
Background: Portal hypertension is the increase in portosystemic pres¬sure gradient in any portion of the portal venous system. The portosystemic gradient is assessed by measuring the wedged hepatic venous pressure (a measure of sinusoidal hepatic pressure) and subtracting the free hepatic venous pressure (systemic pressure) thus obtaining the hepatic venous pressure gradient (HVPG).
Aim of the Work: To describe different patterns of portal hypertensive gastrointestinal vasculopathy in patients with liver cirrhosis. To identify the relationship between these different patterns and the clinical, laboratory and imaging findings in those patients.
Methodology: This cross sectional study was performed at Ain Shams University Hospital and Ahmed Maher Teaching Hospital. Two hundred patients with evidence of liver cirrhosis were recruited consecutively among those attending the outpatient clinics or admitted at the Tropical Medicine Department at Ain Shams University Hospital and Hepatology department at Ahmad Maher Teaching Hospital in the period from May 2014 to January 2016.
Results: This study was conducted in tropical medicine department at Ain Shams university hospital, and hepatology and tropical medicine department at Ahmed Maher teaching hospital. Two hundred patients with liver cirrhosis were recruited in this study.
Conclusion: Large EV are related to more advanced liver disease, lower platelet and WBC count, and high portal hypertension Doppler parameters. Similarly, severe PHG is more prevalent in higher Child score, and is associated with higher portal hypertension and congestion indices, and lower portal vein flow velocity.
Recommendations: More studies are needed on use of the ultrasonographic Doppler as a non-invasive predictor of different portal hypertensive GIT vasculopathy in patients with liver cirrhosis.
Colonoscopy can be advised for cirrhotic patients having large esophageal varices, to detect rectal varices and other portal hypertensive colopathy lesions, which may be a source of lower GI bleeding in those patients.