الفهرس 
INTRODUCTIONOnly 14 pages are availabe for public view
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Abstract
The posterior cruciate ligament (PCL) is an intraarticular extrasynovial structure covered by a thick synovial membrane that receives rich blood supply from a branch of the middle genicular artery, which contributes to its great healing capacity compared to the anterior cruciate ligament (ACL).1 The PCL is considered the primary restraint to posterior tibial translation and a secondary restraint to internal tibial rotation in higher degrees of flexion.2 It is composed of two bundles which were believed to function independently, the larger anterolateral bundle (ALB) predominantly being an important stabilizer in flexion and the smaller posteromedial bundle (PMB) acting in extension.3 However, recent biomechanical studies have found a co-dominant relation between both bundles throughout the whole range of motion.4 The majority of PCL injuries are caused by a posteriorly directed force on the proximal tibia while the knee is flexed. These most often result from „„dashboard‟‟ injuries with the knee in a flexed position or a fall onto a flexed knee with the foot in a plantar-flexed position.5 Sport related PCL injuries may be caused by forced knee hyperextension resulting in isolated PCL tears or more complex injury patterns involving hyperextension and forced varus or valgus angulation resulting in PCL based knee dislocations involving more than one ligament.