الفهرس 
IntroductionOnly 14 pages are availabe for public view
 |  | from | 114 |  |  |
| | | from | 114 | | |
Abstract
Foot DROP deformity is a life changing disability with great impaction on patients’ gait, function and eventually life. The deformity is caused by injury to the extensor fasicles of the sciatic nerve or the common peroneal nerve causing weakness of the extensor compartment of the leg and loss of ankle dorsiflexion.
Different treatment modalities have been used with the gold standard treatment being anterior transfer of the tibialis posterior tendon. However all classic treatment showed devastating complications on long term follow up. This raised the need for a novel treatment and hence nerve transfer was introduced.
Nerve transfer allows early recovery of motor power as regeneration distance is reduced and repair site is brought closer to neuromuscular junction. In addition with low donor nerve morbidity was recorded.
The current study aimed at evaluating the motor power recovery of extensor muscles of the leg following double nerve transfer to the deep peroneal nerve trunk. In principle, the use of two donor nerves provides higher number of crossing axons at repair site so increasing the chance of regeneration. Nerve to LHGNM and nerve to FDL were selected as combined they have an almost similar diameter and cross sectional area as the recipient deep peroneal nerve.
The study included 16 patients with foot DROP deformity due to nerve injury. Examination and evaluation confirmed that patients met the inclusion criteria. After surgery, all patients were followed monthly with a minimum of 12 months follow up after surgery required including the patient’s results in the study. Physiotherapy and rehabilitation focused on
maintaining ankle ROM as well as relearning and re-education modalities.