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العنوان
Ameliorative Effect of Amygdalin on Liver Damage Induced by Erlich Ascites Carcinoma in Mice /
المؤلف
Nassar, Hebatala Abdellatief Mohammed.
هيئة الاعداد
باحث / هبه الله عبد المنصف محمد نصار
مشرف / افراح فتحى سلامة
مناقش / صفوت محمد قاسم
مناقش / محمد حسين محمد حسين
الموضوع
Chemistry.
تاريخ النشر
2021.
عدد الصفحات
170 p. :
اللغة
الإنجليزية
الدرجة
ماجستير
التخصص
Biochemistry
تاريخ الإجازة
16/11/2021
مكان الإجازة
جامعة طنطا - كلية العلوم * - Chemistry
الفهرس
Only 14 pages are availabe for public view

from 47

from 47

Abstract

Cancer is a type of diseases or ailments categorized by uncontrolled division of cells and the aptitude of these cells to spread, either by direct growth into tissue through invasion, or by implantation into distant sites by metastasis where cancer cells are transported over the bloodstream or lymphatic system. Typically, cancer is one of the fatal genetic as well as induced diseases resulting due to the defects in the genetic material. This is because of the loss of cell growth control mechanism owing to numerous carcinogenic mediators for instance physical, chemical and biological agents. These carcinogens lead to reversible as well as irreversible changes in the genetic material and disturb the functionality of cell cycle control proteins. The treatment of cancer comprises surgery, chemotherapy, radiation therapy, hormonal therapy and biological therapy. Consequently, struggles have been made to recognize both natural and synthetic anticancer and antioxidants. In particular, afterward the recommendations of the World Health Organization that studies of antitumor and antioxidants agents of interest has stand up for usage as more effective and safe anticancer and antioxidant agents. Consequently, examinations for effective and non-toxic natural antitumor capability led to increasing and promoting our attention to figure out the influence of the highly antitumor amygdalin, with sorafenib on Ehrlich ascites carcinoma (EAC)–bearing mice and the probability of using them to improve the biochemical changes induced by tumor and their possible role in suppressing the tumor growth rate.