الفهرس 
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Abstract
Primary open-angle glaucoma is a progressive chronic optic neuropathy, typically bilateral. It is a worldwide leading cause of irreversible blindness. Primary open-angle glaucoma corresponds to a progressive loss of retinal ganglion cells characterized by an excavation of the optic disc associated with typical visual field defects. Elevated intraocular pressure is the main risk factor (1). Primary open-angle glaucoma (POAG) is the most prevalent type of glaucoma worldwide (about 70% of the cases of glaucoma) (2). POAG characterized by gonioscopically open anterior chamber angle, with or without elevated intraocular pressure (IOP) which is not associated with known systemic or ocular disorders that affect aqueous outflow and/or cause optic nerve injury (3, 4). The disease is controllable, and the visual impairment caused by glaucoma is irreversible, so early detection is essential. Early diagnosis depends on examination of the optic disc, retinal nerve fiber layer, and visual field (5). Because both sclera and cornea have similar collagen content (6), the biomechanical properties of lamina cribrosa and cornea may be associated. So, the study of corneal biomechanics can indirectly reflex the compression and damage in optic nerve lamina cribrosa (7, 8).