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Abstract December 2019, a series of acute atypical respiratory diseases occurred in Wuhan, China, which rapidly spread from Wuhan to other areas. It was soon discovered that a novel coronavirus was responsible. This novel coronavirus was named severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) due to its high homology (” " ~ " ”70%) to SARS-CoV (Guan et al., 2020). SARS-CoV-2 is a single-stranded positive-sense RNA coronavirus that enters the human cell mainly through binding to angiotensin converting enzyme 2 (ACE2), which is expressed in increased amounts in the alveolar cells of the lung, cardiac myocytes, vascular endothelium, and other cells. SARS-CoV-2 is transmitted mainly after the viral particles are inhaled and enter the respiratory tract. This virus can survive for up to 24–72 hours on surfaces that allow its transmission (Bikdeli et al., 2020). The symptoms of patients infected with SARS-CoV-2 range from minimal symptoms to severe respiratory failure with multiple organ failure. On Computerized tomography (CT) scan, the characteristic pulmonary ground glass opacification can be seen even in asymptomatic patients (Guan et al., 2020). |