الفهرس 
INTRODUCTIONOnly 14 pages are availabe for public view
 |  | from | 172 |  |  |
| | | from | 172 | | |
Abstract
Jaundice may be present at any time during the neonatal period nding on the condition responsible for it. Neonatal jaundice is classified into two groups which include: Early onset jaundice that appears during the first week of life and includes ’J r Rh, ABa incombatibility, physiological jaundice and neonatal ./ , .~ septicaemia. ~. Late onset jaundice that appears after the first week or persists for more than fc :,o 2 weeks. This group is subdivided into unconjugated and conjugated , hyperbilirubinemia. Unconjugated hyperbilirubinemia includes prolonged physiological jaundice, breast milk jaundice, chronic hemolysis. . .The goal of therapy in unconjugated type of jaundice is to prevent the neurotoxicity caused by indirect bilirubin and the main lines adopted are exchange transfusion and phototherapy. .Conjugated hyperbilirubinemia includes systemic viral bacterial or parasitic infections, metabolic disease as a-I antinypsin deficiency, Endocrinal , diseases as hypothyroidism, T.P.N., Choledochal cyst, spontaneous perforation is’ .; J of bile ducts, intrahepatic bile duct paucity, Neonatal hepatitis and biliary ,r ’:; atresia. The last 2 causes make up the majority of cases of cholestasis. Biliary atresia is defined as the obliteration of the extrahepatic and/or the intrahepatic bile ducts, due to an inflammatory process in the perinatal period of unknown aetiology.