الفهرس 
review of literatureOnly 14 pages are availabe for public view
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Abstract
Because pcas is not a discrete disease but rather a spectrum of symptomatology, pathology, and laboratory findings, complete description of its etiology and pathogenesis has not been elucidated. What has evolved is the concept of pcas as a self-perpetuating cycle in which the hypothalamus, pituitary, ovaries and adrenals all contribute to an endocrine imbalance that is usually associated with oligo-ovulation, hirsutism, and infertility (Seibel, 1990). The clinical spectrum is broad and extends from relatively normal menses to chronic oligomenorrhea or amenorrhea and from no hirsutism to virilization. Also the symptoms may range from simple cystic acne, cephalic hair loss, or mild facial hirsutism to instances of oligomenorrhea or amenorrhea, sterility and severe generalized hirsutism (Amer et al., 2002). The diagnosis of polycystic ovarian disease depends on the clinical features, laboratory investigations as gonadotropins, _1l1l9rogens and sex steroid levels in the urine and plasma, also by ultrasonograpy color Doppler, MRI and Laparscopy (Balen 1999). The success of the different line of treatment depends upon the proper selection of patients. Induction of ovulation is either medically or surgically (Campo 1998).