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Abstract Acute myocardial infarction ( MI) is the leading cause of death in North America and Europe. In the united states, the yearly death tool due to coronary heart disease is higher than 500.000. More than 1 million people suffer of acute MI, each year. An additional 200.0000 to 300.000 are estimated to die of acute MI before hospitalization(1). Preinfaction symptom status has long been recognized as an important determinant of clinical outcome after acute MI. Approximately 50% to 60% of patients with acute MI have angina pectoris before the developement of infarction.(2-3) But this ratio varies depending on the study population. The presence of collateral circulation is more common in patients with than without angina.(4-5) Furthermore, the recruitability of these collateral channels during transient coronary occlusion in coronary angioplasty appears to be related to preexisting critical coronary artery stenosis. |